Like every cardiologist on the planet, I used to think oxidized LDL cholesterol created the plaque that caused atherosclerosis (hardening of the arteries). Trouble was, there were many cases of cardiovascular disease that this theory couldn’t account for. Oxidized LDL may be part of the story, but it’s not the full explanation. The exciting news is that nanobacteria may well be. 

Nanobacteria, formally known as Nanobacterium sanguineum, are so minute that they eluded researchers for decades. They’re 1/1,000 the size of normal bacteria, and until recently, nobody believed that something so small could even be alive. It turns out that nanobacteria are not only very much alive but thriving, and they are damaging our health in more ways than we could have ever dreamed. As you know, a mission of mine has been to try to explain how and why heart disease occurs in people who don’t exhibit the traditional risk factors. If we can hit upon the cause, then we can help prevent thousands of unexplained deaths each year. There have been numerous hypotheses, but so many never pan out. Take Chlamydia pneumoniae, the pathogen that causes acute respiratory disease, for example. Remember the news reports from just a few years ago that proclaimed infection with this bacterium probably accounted for much of the unexplained plaque in people? The hope was that we could treat the C. pneumoniae and thereby eradicate the plaque. Well, further research uncovered C. pneumoniae in only a small percentage of all plaque—certainly not enough to be a pervasive cause. I never jumped on the chlamydia bandwagon because the research was not convincing and was too often contradictory. Nanobacteria cause a “false-positive” on tests for Chlamydia. When I first heard about nanobacteria, I was skeptical, too. But I have looked into the subject at length and talked with physicians involved, and now I’m convinced that the study of nanobacteria will bring about a whole new understanding of a number of diseases. This kind of understanding is what leads to treatments and eventually cures.

The NanobacTX Story in Brief: In 1988 Olavi Kajander, M.D., Ph.D. was conducting research on mammalian cells at the University of Kuopio in Finland, work that is ongoing today. As so often happens in basic medical research, the cells in their cell cultures kept dying. But, instead of just throwing them out, as researchers usually do, he had forgotten about the dead cell cultures in the incubator. Months later, he retrieved these cultures and started to investigate an unusual, hardened film that had formed on the culture surfaces. What he discovered were tiny bacteria—20–200 nanometers in size—in calcified shells. The discovery of nanobacteria should have been a major moment in bacterial research history, yet no journal would publish their findings! The bacteria were so small that no one could believe they were alive. It was not until 1997 that their findings were finally published in a medical journal. (This is a good example of how slowly the medical establishment moves.) 

In his research, Dr. Kajander, who has been nominated for a Nobel prize—found that nanobacteria were social little creatures, banding together to secrete an irritating toxic film that causes swelling and inflammation. The film provides them with protection so they can connect and colonize like slime molds, expanding, contracting, and moving. Eventually, this film hardens into a shell, protecting the nanobacteria colony from our body’s attempts to clear it out. Even in this calcified state, nanobacteria aren’t necessarily dormant; they can continue to aggregate and reproduce. And our body doesn’t recognize them as a foreign pathogen at this stage. They’re just seen as calcium, so the nanobacteria are free to expand unchecked. Nanobacteria are a bit sluggish at the multiplication game. Unlike most bacteria, which replicate in minutes or hours, nanobacteria take about three days to reproduce, which helps explain why their significance may have been underestimated for so long. This slow replication means that nanobacteria may be in the body as long as 40 years before symptoms like inflammation and noticeable plaque develop. The biofilm phase of nanobacterial life is one of the most damaging to human bodies, because the biofilm is a potent toxin that causes the body to react powerfully with irritation and swelling. Though the “bugs” themselves cause damage, even more damage is caused by the body’s reaction to them. In other words, the body, in trying to react to the damage, hurts itself. The body’s defenses in turn trigger several medical markers of inflammation, including the well-known C-reactive protein (CRP), which helps explain why elevated CRP levels are a major harbinger of coronary artery disease. 

An Apt Analogy. To help illuminate what the discovery of nanobacteria could ultimately mean for our health, let’s take a look at H. pylori and ulcers. It was only after years of having patients undergo gastric surgery that doctors learned a bacterium known as Helicobacter pylori was the culprit in many ulcers. So surgeons were putting patients with ulcers through major surgery, cutting their vagus nerve (the extensive cranial nerve that extends to the abdomen) and revamping part of their small intestine, when treatment turned out to be easily achieved with antibiotics in most cases. In the same way, interventional cardiologists are going in and cutting the blood vessels around patients’ hearts to bypass plaque-filled arteries in what has become an alarmingly common procedure. We may learn that all that’s needed for severely calcified arteries is a course of the right nanobiotic. (Actually, nanobacteria treatment is more complicated, but I’ll get to that in a moment.) Scientists from the Hungarian Academy of Sciences have reported finding nanobacteria in more than 60 percent of human artery-clogging plaques studied. The Hungarians also confirmed and validated previous research reports of how truly miniscule these bacteria are and therefore how easily they can enter the body via blood exchange and blood products. With their protective apatite coat, nanobacteria are highly resistant to heat, radiation, and most all antibiotics. 

Nanobiotech Pharma, based in Orlando, FL, has been conducting research with NanobacTX, a special therapy that has been instrumental in nanobacteria killing and along with that, plaque reversal. Nanobiotech’s researchers have performed a pilot study of 91 patients using NanobacTX for three months: Of the 91 participating patients, the mean decrease in their coronary artery calcification scores was 58.5% after treatment with NanobacTX therapy for three months. Interestingly, in 19 of those 91 patients 100 percent of coronary artery calcification was eradicated. These are truly amazing statistics! I know of no other treatment that is so successful. Nanobiotech is now conducting a much larger study of plaque reversal, and some of my patients are participating. A minimum of four months of NanobacTX treatment is necessary to treat many folks with coronary artery calcification. For those whose heart plaque burden is excessive, nine to twelve months (or more) of therapy may be needed. From the cardiologists reporting back so far, most patients start seeing a marked improvement in quality of life in four to six weeks. It is also interesting to note that many patients have reported the resolution of co-existing illnesses. (See the list, “Diseases Associated with Calcified Plaque” at the end.)

Despite all the promising early news about NanobacTX, it should not be considered a panacea. For now, I would refer those patients with very high calcium scores or those with angina who have a poor quality of life and are running out of options. This is where the real beauty of NanobacTX treatment lies—providing one more card for us physicians to play when the stakes are the highest. Otherwise, there’s so little we can do when someone’s vessels are clogged beyond repair. Perhaps we’ll eventually discover new combinations of procedures. Cardiologist, James Roberts, MD, FACC, of Toledo, Ohio, reports using NanobacTX as a stepping-stone to EECP.

What You Can Do Now. The bottom line is that coronary artery disease (as well as many other diseases) is a process of inflammation. This inflammation can be caused by nanobacteria and then compounded by traditional risk factors, or it may be the result of any combination of excess oxidized LDL, lipid peroxides from saturated fats, cigarette smoking, heavy metal toxicities, a sedentary lifestyle, and so on. It’s still difficult to say in individual cases what the root cause might be. The science is just not there yet. There are a few blood tests to screen for nanobacteria, but none are available in the US, outside of a research study. I could spend an entire newsletter on this topic, but not every reader would be thrilled. I hope you’re as excited as I am about this breakthrough research. Even as I write this, additional scientific commentary is coming in. This could be the biggest medical science story of the 21st century. 

Nanobacteria has been implicated in the development of: Atherosclerotic Plaque, Blood Disorders, Breast Calcification, Cancer, Cataracts, Glaucoma, Coronary Artery Disease Plaque, Dental Plaque, Eczema, Fibromyalgia, Heart Disease, Kidney Stones, Lichen Planus, Liver Cysts, Multiple Sclerosis (MS), Lou Gehrig’s Disease, Alzheimer’s Disease, Osteoarthritis, Prostatitis, Polycystic Kidney Disease, Psoriasis, Periodental Disease, Gingivitis, BPH, Prostatitis, Prostate Calcification, Rheumatoid Arthritis, Scleroderma and more. Nanobacteria and its pathological calcification are implicated to be either the cause or instrumental in most ALL degenerative disease processes. END OF SINATRA ARTICLE.